E-miR statistics

info info

Citations per year

Number of citations per year for the bioinformatics software tool E-miR

Tool usage distribution map

info info

Associated diseases


Popular tool citations

chevron_left Whole genome alignment File format conversion Adapter trimming Bioinformatics workflows ncRNA identification chevron_right
Want to access the full stats & trends on this tool?


E-miR specifications


Unique identifier OMICS_10682
Name E-miR
Software type Pipeline/Workflow
Interface Command line interface
Restrictions to use None
Biological technology Illumina
Operating system Unix/Linux
Programming languages Perl
Computer skills Advanced
Stability No
Maintained Yes


No version available


  • person_outline Henk Buermans

Publication for E-miR

E-miR citations


Analysis of the microRNA signature in left atrium from patients with valvular heart disease reveals their implications in atrial fibrillation

PLoS One
PMCID: 5933750
PMID: 29723239
DOI: 10.1371/journal.pone.0196666

[…] related disease. As the 299 miRNAs identified in this study matched only partly the 213 miRNAs identified previously in LA tissue (48), only five miRNAs were found commonly altered in both studies (i.e miR-378a-3p, miR-15b-5p, miR-21-5p, miR-125b-5p, and miR-451a). The 42 altered miRNAs were predicted to target cellular pathways known to be involved in AF (i.e. electrical remodeling, and signaling […]


miR 708 5p and miR 34c 5p are involved in nNOS regulation in dystrophic context

PMCID: 5924477
PMID: 29703249
DOI: 10.1186/s13395-018-0161-2

[…] In this study, we used a variety of bioinformatic, molecular, and cell biological methods to demonstrate the role of miRNAs in driving nNOS expression. We selected 4 miRNAs (i.e., miR-31, miR-708, miR-34c, and miR-212) since they were overexpressed in muscular biopsies of BMDd45-55 patients compared to healthy subjects or in muscular biopsies of patients with severe phenoty […]


Comprehensive analysis of blood cells and plasma identifies tissue specific miRNAs as potential novel circulating biomarkers in cattle

BMC Genomics
PMCID: 5894187
PMID: 29636028
DOI: 10.1186/s12864-018-4646-5

[…] There was incomplete overlap between the lists of most abundant miRNAs in plasma and cells, with only 5 of the 15 most abundant miRNAs being common to the two lists, i.e. miR-486, miR-142-5p, miR-191, miR-92a and miR-30e-5p (Table ).Principal component analysis (PCA) of a total of 212 miRNAs that were detected with ≥25 RPMM in more than two samples of either plasma […]


Identification of lncRNA expression profiles and ceRNA analysis in the spinal cord of morphine tolerant rats

PMCID: 5894177
PMID: 29636075
DOI: 10.1186/s13041-018-0365-8

[…] nt study, we predicted that miR-219b, rather than miR-219-5p, would exert this function. Recent research about miR-219 has mainly concentrated on the functions of miR-219-5p, and the other isoform, i.e., miR-219b, has not been studied in detail. However, on the one hand, we identified miR-219-5p and miR-219b, which have both previously been reported to function in the suppression of the proliferat […]


Salivary microRNAs as new molecular markers in cleft lip and palate: a new frontier in molecular medicine

PMCID: 5922367
PMID: 29721173
DOI: 10.18632/oncotarget.24838

[…] //www.targetscan.org/, http://pictar.mdc-berlin.de/, http://www.microrna.org/microrna/home.do) that contain the miR-binding site(s) in the UTR. TargetScan analysis predicted the binding of miR-324-3p e miR-223 to the 3′-UTR of methylenetetrahydrofolate reductase (MTHFR) (Table ). […]


Effects of repeated sprints training on fracture risk associated miRNA

PMCID: 5915055
PMID: 29719588
DOI: 10.18632/oncotarget.24707

[…] ated with fracture risk. The kinetics of response differed: miR-23a-3p, miR-24-3p, and miR-100-5p showed a net decrease by the end of the 8 weeks, while other miRNAs were only transiently modified (i.e., miR-122-5p, miR-125-5p, miR-148a-3p, miR-93-5p). Moreover, contrary to previous findings on cohorts of elderly subjects, some miRNAs associated with bone cell function, i.e., miR-124-3p [–] and mi […]

Want to access the full list of citations?
E-miR institution(s)
Center for Human and Clinical Genetics, Leiden University Medical Center, ZC Leiden, Netherlands
E-miR funding source(s)
This research was supported by the European Union FP6 program HeartRepair LSHM-CT-2205-018630, a Horizon grant (# 93511015) and Centre for Medical Systems Biology within the framework of the Netherlands Genomics Initiative (NGI)/Netherlands Organisation for Scientific Research (NWO).

E-miR reviews

star_border star_border star_border star_border star_border
star star star star star

Be the first to review E-miR